Long -term drinking carbonated drinks can cause gout?Experts say that

In recent years, many young people and even fifteen or six years old have suffered from gout. Why are gout more and more young? Tang Xixi, a national nutrition and health expert and chief physician at the Hunan Provincial Chief Hospital, said in an interview with a reporter today that many young people do not like to drink boiled water, but choose to drink carbonated drinks, but long -term drinking carbonated drinks will greatly increase the risk of gout. Essence

There are many reasons for gout formation, of which the disorders of purine metabolism can cause gout. Purine is an indispensable part of the human body. Under normal circumstances, the amount of purine produced by the human body can maintain a balanced daily, but excessive drinking carbonated drinks will cause purine metabolism disorders, increase the number of purines in the body, and cannot discharge timely in time. in vitro.

Tang Xixi introduced that most of the raw materials of carbonated drinks contain fructose, and fructose can accelerate the degraded purine nucleotide to increase the level of uric acid in the body. Studies have shown that drinking more than 3 cups of drinks a day will increase uric acid value in blood. If gout patients are diabetes concurrently, they cannot drink sugar -containing beverages. In addition, the common fruit juice in life also contains a lot of fruit sugar. Many people think that drinking fruit juice is equivalent to eating fruits, but it is not. Because after the fruits are squeezed into juice, most of the nutrients are removed as the pulp is removed. Only the fruit sugar is released to the greatest extent, which is the end of the house.

How to stay away from gout? Tang Xixi said that reasonable and balanced diet, scientific exercise, reasonable medication, and regular monitoring are an important means to prevent gout.

1. Scientific diet is the foundation

Unreasonable diet is one of the important factors that induce gout. 20%of the human body is exogenous. Therefore, by adjusting the diet structure, uric acid production can be reduced to a certain extent. Specific requirements are as follows:

(1) Limit high purine food

High purine foods such as animal viscera, seafood, thick broth, mushroom vegetables such as vegetables will increase uric acid production, so you should eat as little or not to eat as much as possible. Essence

(2) Stay away from the cup

Because alcohol can interfere with uric acid excretion through the kidneys, beer itself contains a large amount of purine, so gout patients are best to quit alcohol. Whether it is beer, liquor or red wine, try not to drink. In addition, the fructose in beverages also affects the metabolism of uric acid, so the sweet drink does not drink as much as possible.

(3) Drink plenty of water

Drinking more water and urination can help the excretion of uric acid. Even if there is no thirst, sufficient amounts of water should be intake every day. Patients with gout should drink water per day above 2000ml. White boiled water and fresh tea, especially sodium bicarbonate, can be able to be able to, capable The soda of alkaline urine is a good choice.

(4) The body is not super heavy

Research data show that the degree of obesity is directly proportional to the content of blood uric acid. Control weight can help reduce uric acid and prevent gout. Therefore, patients with obesity gout must also "hold their mouths, open their legs", and try to control their weight in the normal range. In addition, excessive exhaustion, severe activities (such as fast running), trauma, cold, etc. can induce acute attacks on gout, and patients with gout should be avoided.

2. Reasonable medication is the key

(1) Urexin drug should not be added during acute seizures

The problem that urgently needs to be solved during the acute seizure of gout is arthritis and swelling and pain. The symptomatic treatment drugs with anti -inflammatory and analgesic effects should be selected (such as non -inflammatory drugs, collateral pastrolasine, glucocorticoids, etc.); and uric acid reducing drugs (hyperteriated drugs, etc. Such as allopurinol, non -tiste, benzromedmine, etc.) itself does not have anti -inflammatory and analgesic effects, which is invalid for controlling arthritis and pain.

In addition, during the acute attack of gout, uric acid reducing acid should not be used, but the symptoms of pain should be completely relieved, and uric acid drugs should be taken after the acute period.

If the patient has started taking uric acid before, it should continue to take it without stopping the drug. The purpose of doing this is to maintain the relatively stable blood uric acid concentration of patients as much as possible to avoid worsening the disease due to significant fluctuations in blood uric acid concentration.

(2) Do not stop uric acid lowering drugs without authorization

Gout is a metabolic disease. At present, it cannot be completely cured. Even if the blood uric acid has dropped to the target value, the drug should not be stopped without authorization. Instead, the drug should be gradually reduced to the minimum effective dose that can maintain blood uric acid within the target range. And adhere to long -term use, the purpose is to enable blood uric acid to stabilize within the target range for a long time.

(3) The rate of uric acid should not be too fast

The gout seizures are not only related to high uric acid, but also have a lot to do with the significant fluctuation of uric acid levels. When the concentration of blood uric acid is reduced suddenly, the uric acid crystals attached to the joints and other places can be quickly dissolved, producing some insoluble needle -shaped crystals. After these microcrystals are swallowed by white blood cells, the inflammatory medium is released, thereby inducing the arthrocascular membrane of the arthroconia film Inflammation and pain. It is recommended to start with a small dose and slowly increase the dose from small doses. When starting uric acid reducing drugs, it is best to combine small doses of anti -inflammatory and analgesic drugs until the stability of blood uric acid is up to standard.

(4) Try to avoid taking drugs that affect uric acid excretion

Such as penicillin, ninosone, thiazide diuretics and rapid urine, pyrdinamide, and ethylene butanol, large -dose aspirin (greater than 2 g per day), etc. These drugs can affect uric acid excretion and increase blood uric acid.

3. Regular monitoring is a guarantee

Regular monitoring of blood uric acid is an important basis for understanding the control of blood uric acid, adjusting diet and medication, and the guarantee of ensuring long -term control of blood uric acid. The governance does not meet the standard, and it is not cured, and uric acid monitoring cannot be relaxed at any time. (All media reporter Li Chuanxin correspondent Chen Siyu Yang Zhengqiang)

[Edit: Wang Yaobing Intern Tang Jiajun] [Source: Hunan Daily · New Hunan Client]

- END -