Ten years of anti -cancer, but died of acute myocardial infarction, doctor: coronary heart disease first aid, grasp two key points

This is a case of a son about his father.

My son is also a patient in our cardiology because of arrhythmia went to the hospital to retract. He could enter the clinic, but talked about his 74 -year -old father.

Just two days ago, his father died of acute myocardial infarction. From the disease to the invalidation of the rescue, 3 hours.

He said that his father felt bad that afternoon, and the "heart" was uncomfortable, so he drove home immediately. Because the unit building has no elevators, on the 6th floor. After arriving at home, call 120 and take it to the hospital for emergency treatment. Checking the electro -image is acute myocardial infarction. Hurry up the green channel and enter the catheter room half an hour.

But at first glance, vascular lesions were too serious. The three main coronary arteries, the left rotation branch and the right crown are completely blocked, and the front decrease was blocked by 97%. The other branches of the other branches were also severe stenosis. Before the doctor had time to expand the blood vessels, the patient had a ventricular fibrillation and the heartbeat stopped. As a result, cardiopulmonary rescue began in the catheter room. Unfortunately, the coronary path lesions are severe, myocardial ischemia is severe, and rescue is invalid.

Speaking of which, the old man who died of myocardial infarction did not diagnose coronary heart disease before, but there was a "heart discomfort" attack in the week before myocardial infarction. There is a history of hypertension, and the tumor has been well controlled because he has taken 10 years of targeted drugs with liver cancer for 10 years.

Many targeted drugs for antitumor can cause hypertension and myocardial damage, myocardial infarction, and Rigffitini is one of them. Let's say this again.

However, no matter what the reason, the coronary heart disease is not ascendant, but sometimes it is ignored. The patient had a history of hypertension. One week before myocardial infarction, angina pectoris symptoms appeared, but there was no medical treatment. This loses the first key time: open blood vessels before the occurrence of myocardial infarction to prevent the occurrence of myocardial infarction. Next, the patient had incorrect help after the myocardial infarction, delayed treatment, exacerbated the deterioration of the condition, and did not grasp the second key timing.

Therefore, we say that the crown heart patient must grasp two key points and choke the sickness.

The first key point, unstable angina pectoris, block the progress of the disease and avoid myocardial infarction. Before the heart infarction of coronary heart disease, it was the stage of unstable angina pectoris. It was previously called "early infarction". Effective treatment during this period can prevent the development of myocardial infarction and effectively protect the function of the heart.

What is unstable angina pectoris?

Except for stable labor angina, others are unstable angina pectoris.

Labor angina pectoris is a angina between a certain physical load. For example, it ’s okay to go flat, and angina pectoris appear on the two or three floors; or if you walk slowly, you ca n’t walk quickly; if you walk hungry and walk, you can n’t do it. What happens during the activity. If such angina pectoris occurs for a period of time, for more than two months, and the degree of attack is relatively stable, it is stable labor angina pectoris. Except for this, other angina pectoris belongs to unstable angina pectoris.

The newly -occurring labor angina pectoris, just like this patient has just happened a week, usually within two months, belongs to the first occurrence; the original labor angina pectoris is worsened, which is deteriorating; When it happens, the lying position of the position; the mutant angina pectoris caused by coronary spasm, these belongs to the unstable angina pectoris.

Everyone knows that blood vessels can relax and shrink, blood vessels relax, and blood flow will increase. For coronary arteries, this is the reserve function, which can increase blood supply when needed. However, after arteriosclerosis, the blood vessels have poor diastolic capacity, and the reserve ability decreases. Once the blood supply is needed, it will not be supplied, and the myocardial ischemia will be ischemia. Generally speaking, coronary heart disease patients have angina pectoris, and coronary stenosis is more than 50%. The clinical is more than 75%. If according to the probability of pre -verification, if elderly men have a typical symptom of angina pectoris, the diagnosis of coronary heart disease is not to leave. Therefore, a new occurrence of angina pectoris or worsening condition must go to the hospital in time.

The second key point is that when acute myocardial infarction occurs, seek help in time, and as soon as possible as soon as possible. Of course, patients cannot be determined when the patient is ill. However, myocardial infarction, myocardial ischemia and necrosis, the symptoms of patients will be severe, the degree of uncomfortableness will be more serious, weak, sweating is often, some people still have nausea and vomiting, and there is a convenient. At this time, the judgment is not clear, so it is dealt with according to the most serious heart disease.

Do not continue to move, rest quietly, call for emergency treatment, and go to the hospital. Like the patient drove home by himself, he also went to 6 floors. He did not say the danger, and delayed the treatment time. In the early stage of myocardial infarction, in the early stages of myocardial ischemia and necrosis, biological electricity would be disordered, and ventricular fibrillation was most likely to occur. The patient eventually stopped the arrest of ventricular fibrillation.

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